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Tuberculosis diagnosis remains challenging, particularly distinguishing between active and latent infection. Misdiagnosis drives unnecessary antibiotics use, and disproportionately affects HIV-positive patients who have the greatest clinical need for accuracy.

Stanford researchers have identified a small set of genes that can be used to diagnose active tuberculosis (TB), distinguish active TB from latent TB or other diseases, and predict progression from latent to active TB months before conventional tests.

Stanford researchers have developed a powerful immune-based gene expression signature that accurately predicts severe outcomes and all-cause mortality across high-risk populations, enabling personalized treatment selection and advancing precision medicine in critical illnesses

Stanford researchers have developed a method of reducing pulmonary hypertension (PH) in mammals by targeting FHIT (Fragile Histidine Triad), a gene not previously linked to PH but consistently reduced in blood of patients with pulmonary arterial hypertension (PAH).

Researchers at Stanford have developed a gene expression-based method for determining a virally infected patient's risk of developing severe symptoms, irrespective of the virus.