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Researchers at Stanford have identified why engineered pathways that convert formate into multi-carbon products chronically underperform, and have developed an enzymatic repair strategy that recovers the inactivated enzymes, increasing pathway flux and extending reaction longe

Huntington's Disease and other ataxias are devastating diseases without any cure or treatment. They are caused by the formation of toxic oligomeric and the aggregation of the Huntintin (HTT) protein.

Researchers at Stanford have developed genetically encoded STING agonists that enable controlled activation of the STING pathway from within engineered cells.

Researchers at Stanford have developed constitutively active, programmable synthetic cytokine receptors capable of polarizing human macrophages to a variety of user-defined cell states which may be useful for therapeutic applications.

Stanford researchers have engineered a heterologous system for the high-efficiency production of numerous taxanes and taxane intermediates.

Stanford researchers have developed a method using trained learning models to optimize synthetic DNA libraries for high-throughput molecular biology experiments.

Fast, accurate and cheap synthesis of ultralong strands of DNA is an essential foundation for synthetic DNA technologies such as cellular programming and engineering.

Dr. Mark Kay and colleagues have created antibiotic-selectable, non-silencing plasmid vectors that can be prepared by conventional methods and provide persistent high levels of transgene expression.