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SARS-CoV2 is known to gain entry into epithelial cells through the association of its viral spike protein with the ACE2 receptor, which is widely expressed on epithelial cell types.

Stanford scientists have discovered that a specific protein signaling pathway can promote regenerative wound healing by suppressing fibrosis-related mechanosignaling.

Researchers at Stanford University have identified EP300 modulators as effective treatments for reducing skin scarring, demonstrating significant regeneration of hair follicles, sweat glands, and connective tissue architecture.

Researchers at Stanford University have developed a novel therapeutic avenue for the treatment of osteoporosis and other musculoskeletal diseases.

Researchers at Stanford University have found that recombinant osteopontin (SPP1) protein reduces foreign body response (FBR) and thereby facilitates successful integration and function of implantable devices.

Stanford scientists have developed a novel hydrogel for long-term drug delivery of an Activator Protein 1 (AP-1) inhibitor for the prevention of post-surgical abdominal adhesion.

The Longaker lab at Stanford University has developed a machine learning algorithm that can analyze and detect fibrotic disease.

The Longaker lab at Stanford University has recently discovered that local injection of the drug Verteporfin after wounding can reduce scarring, improve the strength of healed skin, and regrow the hair follicles and sweat glands that are usually lost during the scarring proces

Skin wounds invariably heal by developing fibrotic scar tissue, which can result in devastating disfigurement, growth restriction and permanent functional loss.

Researchers at Stanford have proposed that coating surgical sutures with mechanotransduction inhibitors may reduce scarring of the tissue.

Stanford researchers have invented a novel nanobody-based platform for specific activation of bone and cartilage regeneration.

Stanford researchers have found that aging and disease lead to skeletal stem cell (SSC) dysfunction and hence decreased bone health.

Researchers at Stanford have identified a novel method of use for the drug T-5224 in the prevention of abdominal adhesion.

Radiation therapy is a common option in diseases like breast cancer, but can also cause significant damage to the skin. Permanent scarring and fibrosis can result, with both aesthetic and functional consequences for cancer patients.

Researchers at Stanford have developed methods to enhance bone healing in diabetic patients, who often suffer from impaired fracture healing due to a deficiency in hedgehog signaling in their skeletal stem cells.