Stanford researchers have developed a novel gene therapy vector, AAV-capGL to overcome immune barriers that currently limit the efficacy and safety of adeno-associated virus (AAV)-based gene therapies.
Stanford researchers have developed a fast and flexible platform for building human brain organoids that mimic the complexity of the brain's cellular makeup. This breakthrough enables faster research and better disease modeling for neurological conditions.
Researchers at Stanford have developed a clinically applicable method of bone marrow conditioning for stem cell transplantation or treatment of hematologic malignancies.
High-grade gliomas, including glioblastoma and diffuse midline glioma are the most common malignant brain tumor types and leading causes of brain-tumor-related death in adults and children, respectively.
Stanford researchers have developed a novel CRISPR-based method, Oligo-LiveFISH, for generating large-scale pools of synthetic RNA oligos that enable multiplexed targeting, imaging, and manipulation of genomic regions in living cells.
Stanford scientists have developed a frequency-based power control method that enables RF amplifiers to double their output power within 500 nanoseconds using only passive components.
Researchers at Stanford have developed a novel T cell engineering platform that leverages constitutively active interleukin-9 receptor (IL-9R) signaling to improve the efficacy and scalability of immunotherapies for solid tumors.
Stanford scientists have developed a new DNA-based technology that allows therapeutic genes to be maintained in human cells for extended periods without altering the cell's chromosomes.
Stanford researchers have developed the Large-scale Electrophysiology Amplification Platform (LEAP), a wireless, label-free optical system for monitoring the electrical activity of neurons and heart cells.
Stanford researchers have developed a novel, multi-specific chimeric antigen receptor (CAR) T-cell therapy designed to overcome the key challenges of treating solid tumors, including tumor heterogeneity, immune evasion, and CAR T-cell exhaustion.
Stanford scientists have discovered that a specific protein signaling pathway can promote regenerative wound healing by suppressing fibrosis-related mechanosignaling.
Lithium thionyl chloride batteries are one of the most energy dense batteries but have attracted limited prior interest due to their lack of rechargeability.